Interrupted Memories: Alcohol-Induced Blackouts National Institute on Alcohol Abuse and Alcoholism NIAAA

Alcohol Blackouts

Researchers have largely ignored the occurrence of blackouts among young social drinkers, so the idea that blackouts are an unlikely consequence of heavy drinking in nonalcoholics has remained deeply entrenched in both the scientific and popular cultures. Yet there is clear evidence that blackouts do occur among social drinkers. Knight and colleagues (1999) observed that 35 percent of trainees in a large pediatric residency program had experienced at least one blackout.

  1. The brain’s ability to create long-term memories is completely blocked.
  2. A blackout is not the same as “passing out,” which means either falling asleep or losing consciousness from drinking too much.
  3. Manipulations that disrupt the theta rhythm also disrupt the ability to perform tasks that depend on the hippocampus (Givens et al. 2000).
  4. Studies seem to agree that heavy drinking alone doesn’t cause blackouts.
  5. Timmen L. Cermak, MD, is a psychiatrist who specializes in addiction medicine.

All but two kept their hand up, and one who had lowered his hand said he was adopted and did not know about his parents. One of the key requirements for the establishment of LTP in the hippocampus is that a type of signal receptor known as the NMDA2 receptor becomes activated. Activation of the NMDA receptor allows calcium to enter the cell, which sets off a chain of events leading to long-lasting changes in the cell’s structure or function, or both. Alcohol interferes with the activation of the NMDA receptor, thereby preventing the influx of calcium and the changes that follow (Swartzwelder et al. 1995). This is believed to be the primary mechanism underlying the effects of alcohol on LTP, though other transmitter systems probably are also involved (Schummers and Browning 2001). We’ve compiled some of the most frequently asked questions about alcohol blackouts here.

Addiction Questions?

Alcohol Blackouts

Because blackouts tend to occur at high BACs, they commonly stem from binge drinking, defined as a pattern of drinking that increases a person’s BAC to 0.08 percent or higher. This typically occurs after 4 drinks for women and 5 drinks for men—in about 2 hours. In fact, many people who have blackouts do so after engaging in a behavior known as high-intensity drinking, which is defined as drinking at levels that are at least twice as high as the binge-drinking thresholds for women and men. For most people, binge-drinking large amounts of alcohol causes them to black out.

You Can’t Function at All During an Alcohol Blackout

It can be hard to identify a blackout in someone else because they may be able to function fairly well. They may drive cars, have full conversations, or engage in sexual acts—all without remembering. Heavy alcohol use contributes to a shrinkage of the brain similar to Alzheimer’s disease, which is characterized by memory loss. However, studies do not support alcohol as a cause of Alzheimer’s disease. Schuckit’s study and several others have found that people who black out from drinking risk a number of negative consequences. People who are experiencing being blackout drunk often feel similar symptoms to being drunk.

During the 1970s, researchers hypothesized that alcohol depressed neural activity by altering the movement of key molecules (in particular, lipids) in nerve cell membranes. This change then led to alterations in the activity of proteins, including those that influence communication between neurons by controlling the passage of positively or negatively charged atoms (i.e., ions) through cell membranes (e.g., Chin and Goldstein 1977). In some cases, only a few amino acids appear to distinguish receptors that are sensitive to alcohol from those that are not (Peoples and Stewart 2000). It remains unclear exactly how alcohol interacts with receptors to alter their activity. However, long-term effects of chronic alcohol abuse — such as liver damage, nerve damage and increased cancer risk — do not always go away.

Perhaps the most common myth about a blackout is that it involves passing out. This might well happen at some point, but during a blackout the person is often still able to talk and laugh and flirt and sing and dance, and may appear to be in control of all their faculties. However, the next day there will be no memory of those things, so it’s as if they didn’t really happen.

Alcohol Blackouts

Are Some People More Likely Than Others to Experience Blackouts?

Damage to the frontal lobes leads to profound cognitive impairments, one of which is a difficulty forming new memories. When a person overconsumes alcohol and blacks out drunk, the hippocampus—the area responsible for learning and memory—in the brain no longer functions properly. Drinking to the point of a blackout has gained pop culture notoriety in recent years. Alcohol-induced blackouts can lead to impaired memory of events that transpired while intoxicated, and a drastically increased risk of injuries and other harms. They can occur in anyone who drinks alcohol, no matter their age or level whippets balloons of experience with drinking.

They feel carefree, are overly friendly or overly aggressive, have slurred speech and can’t walk straight. Signs that they may be blacking out or are blackout drunk include losing their train of thought, not understanding normal speech and getting confused about recent events. A blackout is a loss of the ability to make memories, but people are still conscious when they’re blackout drunk. But we all know that as blood alcohol content goes up, our judgment and coordination go down.

Women who drank until they passed out were hard as nails, one of the boys, and not to be messed with. I was reckless and stupid, but in my mind I was adventurous and exciting. Timmen L. Cermak, MD, is a psychiatrist who specializes in addiction medicine.

Blackouts are much more common among social drinkers than previously assumed and should be viewed as a potential consequence of acute intoxication regardless of age or whether one is clinically dependent upon alcohol. Blackouts represent episodes of amnesia, difference between aa and na during which subjects are capable of participating even in salient, emotionally charged events—as well as more mundane events—that they later cannot remember (Goodwin 1995). Like milder alcohol-induced memory impairments, these periods of amnesia are primarily “anterograde,” meaning that alcohol impairs the ability to form new memories while the person is intoxicated, but does not typically erase memories formed before intoxication.

Intoxicated subjects are typically able to recall information immediately after it is presented and even keep it active in short-term memory for 1 minute or more if they are not distracted. Subjects also are normally able to recall long-term memories formed before they became intoxicated; however, beginning with just one or two drinks, subjects begin to show impairments in the ability to transfer information into long-term storage. Under some circumstances, alcohol can impact this process so severely that, once sober again, subjects are unable to recall critical elements of events, or even entire events, that occurred while they were intoxicated. That’s because the brain’s ability to create long-term memories isn’t affected as much by blood alcohol content as it is by rapid rises in that level. Binge drinking — consuming numerous drinks in a short period— is more likely to cause alcohol blackouts, amnesia and memory loss than slow, heavy drinking, according to numerous studies. As detailed in this brief review, alcohol can have a dramatic impact on memory.

What to Do When Someone Is Too Drunk

With a personalized treatment, you or someone you care about can take the necessary steps to understand and overcome alcohol addiction through residential, partial hospitalization or outpatient programs, depending on your needs. Blackouts may not cause direct harm, but the amount of drinking you have to do to become blackout drunk has serious consequences — especially over time. The more times you blackout, the more times you are impairing your balance, motor skills, impulse control and decision-making to the point where you may do something dangerous or self-injurious. They do sometimes occur in conjunction, but for the most part, people who are experiencing a blackout can still move around and may generally act like they’re having a great time. However, their judgment and decision-making are severely impaired even if they don’t realize it, making it difficult to convince the person not to engage in risky behavior. A standard drink ⁠— that’s 1.5 oz of hard liquor, 5 oz of wine or 12 oz of beer ⁠— will elevate the average person’s BAC by around 0.06 per drink.

As long as excessive drinking continues, these immediate symptoms may extend into chronic mental health conditions. During a blackout, the mind is not functioning properly because the brain is not functioning properly. The fundamental mental dysfunction is that short-term memory is no longer being uploaded duloxetine withdrawal timeline into longer-term memory. Short-term memory is sometimes called scratchpad memory—it records events for only about three minutes before they fade.

He still was able to learn basic motor skills, keep information active in short-term memory for a few seconds or more if left undistracted, and remember episodes of his life from long ago, but he was unable to form new long-term memories for facts and events. The pattern of H.M.’s impairments also forced a re-examination of models of long-term memory storage. Was able to retrieve long-term memories formed roughly a year or more before his surgery, he could not recall events that transpired within the year preceding his surgery. This strongly suggests that the transfer of information into long-term storage actually takes place over several years, with the hippocampus being necessary for its retrieval for the first year or so.

Subjects were asked to recall details regarding these stimuli 2 minutes, 30 minutes, and 24 hours after the stimuli were shown. Half of the subjects reported no recall for the stimuli or their presentation 30 minutes and 24 hours after the events, though most seemed to recall the stimuli 2 minutes after presentation. Lack of recall for the events 24 hours later, while sober, represents clear experimental evidence for the occurrence of blackouts.

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